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Huntington’s alone is exciting enough,” said Hardy, who first proposed that amyloid proteins play a central role in Alzheimer’s. “I don’t want to overstate this too much, but if it works for one, why can’t it work for a lot of them? I am very, very excited.” Prof Giovanna Mallucci, associate director of UK Dementia Research Institute at the University of Cambridge, described the work as a “tremendous step forward” for individuals with Huntington’s disease and their families. “Clearly, there will be much interest into whether can be applied to the treatment of other neurodegenerative diseases, like Alzheimer’s,” she added. However, she said that in the case of most other disorders the genetic causes are complex and less well understood, making them potentially harder to target. About 10,000 people in the UK have the condition and about 25,000 are at risk. Most people with Huntington’s inherited the gene from a parent, but about one in five patients have no family history of the disease.